strains nctc 11 638 Search Results


95
ATCC e coli nctc 13353
Expanded gene content of 13 ENSE isolates with cefepime/taniborbactam MICs ≥ 4 mg/L
E Coli Nctc 13353, supplied by ATCC, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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94
ATCC reference strains 86 5443 2 2 etbf strain l l myers nctc 9343 ntbf strain atcc
Expanded gene content of 13 ENSE isolates with cefepime/taniborbactam MICs ≥ 4 mg/L
Reference Strains 86 5443 2 2 Etbf Strain L L Myers Nctc 9343 Ntbf Strain Atcc, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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94
ATCC p aeruginosa strains
MIC characterization of the <t> P. aeruginosa </t> isolates used in the experiments
P Aeruginosa Strains, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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95
ATCC c7 s tox
MIC characterization of the <t> P. aeruginosa </t> isolates used in the experiments
C7 S Tox, supplied by ATCC, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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99
ATCC vaca positive strain nctc 11 637
H. pylori transcriptionally increased gastric epithelial NFIL3 expression via ERK-SP1 pathway. ( A ) AGS cells were subjected to infection with H. pylori 11,637 or 26,695 (MOI = 100, 24 h) either in the same (lower) chamber or in a separate (upper) chamber of a Transwell apparatus. The expression levels of NFIL3 were assessed using qRT-PCR ( n = 3) and WB. ( B ) The expression of NFIL3 in AGS cells infected with H. pylori TN2GF4 or its virulence mutant strains was evaluated through the GEO database ( GSE60661 ). ( C ) NFIL3 expression was further examined in AGS cells infected with H. pylori 11,637 or the Δ cagA mutant, utilizing qRT-PCR ( n = 3) and WB. ( D ) The expression of NFIL3 was also analyzed in AGS cells infected with H. pylori 26,695 or the Δ <t>vacA</t> mutant through qRT-PCR ( n = 3) and WB. ( E ) The expression levels of NR1D1 were investigated in AGS cells infected with H. pylori TN2GF4 or virulence mutant strains utilizing the GEO database ( GSE60661 ). ( F ) Potential transcription factor binding sites were predicted via the PROMO website, focusing on a 2000 bp segment of the NFIL3 promoter. ( G ) A conserved sequence of putative SP1 binding sites (red line) was identified in both human and mouse models. ( H ) WB and qRT-PCR ( n = 3) analyses of NFIL3 were conducted in AGS cells post-treatment with H. pylori 11,637, U0126, and Mithramycin A (MMA). ( I ) Following a 24-hour transfection with SP1 siRNA, AGS cells were subsequently infected with H. pylori 11,637 for an additional 24 h. The expression levels of NFIL3 and SP1 were analyzed using qRT-PCR ( n = 3) and WB. ( J ) NFIL3 promoter luciferase reporter assays were conducted in AGS cell lines treated with H. pylori 11,637, U0126, Mithramycin A (MMA), and the Δ cagA mutant. ( K ) Luciferase reporter assay for NFIL3-Luc and mutant-Luc. n.s. P > 0.05, ** P < 0.01, and *** P < 0.001
Vaca Positive Strain Nctc 11 637, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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94
ATCC standard strain v cholerae
H. pylori transcriptionally increased gastric epithelial NFIL3 expression via ERK-SP1 pathway. ( A ) AGS cells were subjected to infection with H. pylori 11,637 or 26,695 (MOI = 100, 24 h) either in the same (lower) chamber or in a separate (upper) chamber of a Transwell apparatus. The expression levels of NFIL3 were assessed using qRT-PCR ( n = 3) and WB. ( B ) The expression of NFIL3 in AGS cells infected with H. pylori TN2GF4 or its virulence mutant strains was evaluated through the GEO database ( GSE60661 ). ( C ) NFIL3 expression was further examined in AGS cells infected with H. pylori 11,637 or the Δ cagA mutant, utilizing qRT-PCR ( n = 3) and WB. ( D ) The expression of NFIL3 was also analyzed in AGS cells infected with H. pylori 26,695 or the Δ <t>vacA</t> mutant through qRT-PCR ( n = 3) and WB. ( E ) The expression levels of NR1D1 were investigated in AGS cells infected with H. pylori TN2GF4 or virulence mutant strains utilizing the GEO database ( GSE60661 ). ( F ) Potential transcription factor binding sites were predicted via the PROMO website, focusing on a 2000 bp segment of the NFIL3 promoter. ( G ) A conserved sequence of putative SP1 binding sites (red line) was identified in both human and mouse models. ( H ) WB and qRT-PCR ( n = 3) analyses of NFIL3 were conducted in AGS cells post-treatment with H. pylori 11,637, U0126, and Mithramycin A (MMA). ( I ) Following a 24-hour transfection with SP1 siRNA, AGS cells were subsequently infected with H. pylori 11,637 for an additional 24 h. The expression levels of NFIL3 and SP1 were analyzed using qRT-PCR ( n = 3) and WB. ( J ) NFIL3 promoter luciferase reporter assays were conducted in AGS cell lines treated with H. pylori 11,637, U0126, Mithramycin A (MMA), and the Δ cagA mutant. ( K ) Luciferase reporter assay for NFIL3-Luc and mutant-Luc. n.s. P > 0.05, ** P < 0.01, and *** P < 0.001
Standard Strain V Cholerae, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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94
ATCC mycoplasma sp strain ovine caprine group 11
H. pylori transcriptionally increased gastric epithelial NFIL3 expression via ERK-SP1 pathway. ( A ) AGS cells were subjected to infection with H. pylori 11,637 or 26,695 (MOI = 100, 24 h) either in the same (lower) chamber or in a separate (upper) chamber of a Transwell apparatus. The expression levels of NFIL3 were assessed using qRT-PCR ( n = 3) and WB. ( B ) The expression of NFIL3 in AGS cells infected with H. pylori TN2GF4 or its virulence mutant strains was evaluated through the GEO database ( GSE60661 ). ( C ) NFIL3 expression was further examined in AGS cells infected with H. pylori 11,637 or the Δ cagA mutant, utilizing qRT-PCR ( n = 3) and WB. ( D ) The expression of NFIL3 was also analyzed in AGS cells infected with H. pylori 26,695 or the Δ <t>vacA</t> mutant through qRT-PCR ( n = 3) and WB. ( E ) The expression levels of NR1D1 were investigated in AGS cells infected with H. pylori TN2GF4 or virulence mutant strains utilizing the GEO database ( GSE60661 ). ( F ) Potential transcription factor binding sites were predicted via the PROMO website, focusing on a 2000 bp segment of the NFIL3 promoter. ( G ) A conserved sequence of putative SP1 binding sites (red line) was identified in both human and mouse models. ( H ) WB and qRT-PCR ( n = 3) analyses of NFIL3 were conducted in AGS cells post-treatment with H. pylori 11,637, U0126, and Mithramycin A (MMA). ( I ) Following a 24-hour transfection with SP1 siRNA, AGS cells were subsequently infected with H. pylori 11,637 for an additional 24 h. The expression levels of NFIL3 and SP1 were analyzed using qRT-PCR ( n = 3) and WB. ( J ) NFIL3 promoter luciferase reporter assays were conducted in AGS cell lines treated with H. pylori 11,637, U0126, Mithramycin A (MMA), and the Δ cagA mutant. ( K ) Luciferase reporter assay for NFIL3-Luc and mutant-Luc. n.s. P > 0.05, ** P < 0.01, and *** P < 0.001
Mycoplasma Sp Strain Ovine Caprine Group 11, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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90
ATCC a hominis
<t> A. hominis </t> isolates examined in this study ( n = 46)
A Hominis, supplied by ATCC, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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96
ATCC q8a9m2 xylose isomerase os bacteroides thetaiotaomicron
<t> A. hominis </t> isolates examined in this study ( n = 46)
Q8a9m2 Xylose Isomerase Os Bacteroides Thetaiotaomicron, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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97
ATCC a6l792 xylose isomerase os bacteroides vulgatus
<t> A. hominis </t> isolates examined in this study ( n = 46)
A6l792 Xylose Isomerase Os Bacteroides Vulgatus, supplied by ATCC, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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94
ATCC q7whb6 chaperone protein clpb os bordetella bronchiseptica
<t> A. hominis </t> isolates examined in this study ( n = 46)
Q7whb6 Chaperone Protein Clpb Os Bordetella Bronchiseptica, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Expanded gene content of 13 ENSE isolates with cefepime/taniborbactam MICs ≥ 4 mg/L

Journal: JAC-Antimicrobial Resistance

Article Title: Activity of cefepime/taniborbactam and comparators against whole genome sequenced ertapenem-non-susceptible Enterobacterales clinical isolates: CANWARD 2007–19

doi: 10.1093/jacamr/dlab197

Figure Lengend Snippet: Expanded gene content of 13 ENSE isolates with cefepime/taniborbactam MICs ≥ 4 mg/L

Article Snippet: Quality control was assured using E. coli ATCC 25922, Pseudomonas aeruginosa ATCC 27853, E. coli NCTC 13353 (CTX-M-15) (routine quality control strain used for cefepime/taniborbactam) and K. pneumoniae ATCC BAA-1705 (KPC-2, TEM, SHV).

Techniques:

Cefepime/taniborbactam and comparator MIC distributions for 16 ENSE isolates with carbapenemase genes

Journal: JAC-Antimicrobial Resistance

Article Title: Activity of cefepime/taniborbactam and comparators against whole genome sequenced ertapenem-non-susceptible Enterobacterales clinical isolates: CANWARD 2007–19

doi: 10.1093/jacamr/dlab197

Figure Lengend Snippet: Cefepime/taniborbactam and comparator MIC distributions for 16 ENSE isolates with carbapenemase genes

Article Snippet: Quality control was assured using E. coli ATCC 25922, Pseudomonas aeruginosa ATCC 27853, E. coli NCTC 13353 (CTX-M-15) (routine quality control strain used for cefepime/taniborbactam) and K. pneumoniae ATCC BAA-1705 (KPC-2, TEM, SHV).

Techniques:

Cefepime/taniborbactam and comparator MIC distributions for 33 ENSE isolates with ESBL genes but no carbapenemase genes

Journal: JAC-Antimicrobial Resistance

Article Title: Activity of cefepime/taniborbactam and comparators against whole genome sequenced ertapenem-non-susceptible Enterobacterales clinical isolates: CANWARD 2007–19

doi: 10.1093/jacamr/dlab197

Figure Lengend Snippet: Cefepime/taniborbactam and comparator MIC distributions for 33 ENSE isolates with ESBL genes but no carbapenemase genes

Article Snippet: Quality control was assured using E. coli ATCC 25922, Pseudomonas aeruginosa ATCC 27853, E. coli NCTC 13353 (CTX-M-15) (routine quality control strain used for cefepime/taniborbactam) and K. pneumoniae ATCC BAA-1705 (KPC-2, TEM, SHV).

Techniques:

Cefepime/taniborbactam and comparator MIC distributions for 139 ENSE isolates with AmpC/Class C β-lactamase genes but no carbapenemase genes

Journal: JAC-Antimicrobial Resistance

Article Title: Activity of cefepime/taniborbactam and comparators against whole genome sequenced ertapenem-non-susceptible Enterobacterales clinical isolates: CANWARD 2007–19

doi: 10.1093/jacamr/dlab197

Figure Lengend Snippet: Cefepime/taniborbactam and comparator MIC distributions for 139 ENSE isolates with AmpC/Class C β-lactamase genes but no carbapenemase genes

Article Snippet: Quality control was assured using E. coli ATCC 25922, Pseudomonas aeruginosa ATCC 27853, E. coli NCTC 13353 (CTX-M-15) (routine quality control strain used for cefepime/taniborbactam) and K. pneumoniae ATCC BAA-1705 (KPC-2, TEM, SHV).

Techniques:

Cefepime/taniborbactam and comparator MIC distributions for 22 ENSE with OXA-family genes but no carbapenemase genes

Journal: JAC-Antimicrobial Resistance

Article Title: Activity of cefepime/taniborbactam and comparators against whole genome sequenced ertapenem-non-susceptible Enterobacterales clinical isolates: CANWARD 2007–19

doi: 10.1093/jacamr/dlab197

Figure Lengend Snippet: Cefepime/taniborbactam and comparator MIC distributions for 22 ENSE with OXA-family genes but no carbapenemase genes

Article Snippet: Quality control was assured using E. coli ATCC 25922, Pseudomonas aeruginosa ATCC 27853, E. coli NCTC 13353 (CTX-M-15) (routine quality control strain used for cefepime/taniborbactam) and K. pneumoniae ATCC BAA-1705 (KPC-2, TEM, SHV).

Techniques:

MIC characterization of the  P. aeruginosa  isolates used in the experiments

Journal: Antimicrobial Agents and Chemotherapy

Article Title: Pharmacokinetics and Pharmacodynamics of Murepavadin in Neutropenic Mouse Models

doi: 10.1128/AAC.01699-18

Figure Lengend Snippet: MIC characterization of the P. aeruginosa isolates used in the experiments

Article Snippet: A dose range of 0.25 to 32 mg/kg was used 1, 2, 4, or 8 times in 24 h. Dose-response experiments were performed for an additional 13 P. aeruginosa strains (5, 6, 9, 11, 12, 15, 16, 19, 21, 22, X11045, ATCC BAA 2113, and NCTC 13437).

Techniques:

Relationships of murepavadin 24-h AUC/MIC (A to C), Cmax/MIC (D to F), and the percentage of time above the MIC (%T>MIC) (G to I) for P. aeruginosa ATCC 27853 in the neutropenic thigh and lung infection models with change in CFU per thigh or lung from start of treatment and after 24 h of therapy. Each symbol represents a therapy response in one mouse thigh or lung. The line is the best-fit line based on the sigmoid Emax model.

Journal: Antimicrobial Agents and Chemotherapy

Article Title: Pharmacokinetics and Pharmacodynamics of Murepavadin in Neutropenic Mouse Models

doi: 10.1128/AAC.01699-18

Figure Lengend Snippet: Relationships of murepavadin 24-h AUC/MIC (A to C), Cmax/MIC (D to F), and the percentage of time above the MIC (%T>MIC) (G to I) for P. aeruginosa ATCC 27853 in the neutropenic thigh and lung infection models with change in CFU per thigh or lung from start of treatment and after 24 h of therapy. Each symbol represents a therapy response in one mouse thigh or lung. The line is the best-fit line based on the sigmoid Emax model.

Article Snippet: A dose range of 0.25 to 32 mg/kg was used 1, 2, 4, or 8 times in 24 h. Dose-response experiments were performed for an additional 13 P. aeruginosa strains (5, 6, 9, 11, 12, 15, 16, 19, 21, 22, X11045, ATCC BAA 2113, and NCTC 13437).

Techniques: Infection

In vivo efficacies of murepavadin against  P. aeruginosa  isolates in the neutropenic lung infection models

Journal: Antimicrobial Agents and Chemotherapy

Article Title: Pharmacokinetics and Pharmacodynamics of Murepavadin in Neutropenic Mouse Models

doi: 10.1128/AAC.01699-18

Figure Lengend Snippet: In vivo efficacies of murepavadin against P. aeruginosa isolates in the neutropenic lung infection models

Article Snippet: A dose range of 0.25 to 32 mg/kg was used 1, 2, 4, or 8 times in 24 h. Dose-response experiments were performed for an additional 13 P. aeruginosa strains (5, 6, 9, 11, 12, 15, 16, 19, 21, 22, X11045, ATCC BAA 2113, and NCTC 13437).

Techniques: In Vivo, Infection

In vivo dose response of murepavadin against three P. aeruginosa isolates in the neutropenic murine lung infection model. (A) P. aeruginosa ATCC 27853. (B) NCTC 13437. (C) Isolate 16. Each symbol represents a therapy response in one mouse lung. The x axis is the murepavadin exposure expressed as AUC of the unbound fraction of the drug from 0 to 24 h (fAUC0–24 h). The y axis is the change in log10 of bacterial burden from the start of treatment. (A) The results from the study conducted at Radboud University are indicated with a blue triangle, and those from the study conducted at Evotec (UK) Ltd. are indicated with a black dot. The ED50 represents the AUC associated with 50% of the maximal effect (Emax). The line is the best fit based on the sigmoid Emax model.

Journal: Antimicrobial Agents and Chemotherapy

Article Title: Pharmacokinetics and Pharmacodynamics of Murepavadin in Neutropenic Mouse Models

doi: 10.1128/AAC.01699-18

Figure Lengend Snippet: In vivo dose response of murepavadin against three P. aeruginosa isolates in the neutropenic murine lung infection model. (A) P. aeruginosa ATCC 27853. (B) NCTC 13437. (C) Isolate 16. Each symbol represents a therapy response in one mouse lung. The x axis is the murepavadin exposure expressed as AUC of the unbound fraction of the drug from 0 to 24 h (fAUC0–24 h). The y axis is the change in log10 of bacterial burden from the start of treatment. (A) The results from the study conducted at Radboud University are indicated with a blue triangle, and those from the study conducted at Evotec (UK) Ltd. are indicated with a black dot. The ED50 represents the AUC associated with 50% of the maximal effect (Emax). The line is the best fit based on the sigmoid Emax model.

Article Snippet: A dose range of 0.25 to 32 mg/kg was used 1, 2, 4, or 8 times in 24 h. Dose-response experiments were performed for an additional 13 P. aeruginosa strains (5, 6, 9, 11, 12, 15, 16, 19, 21, 22, X11045, ATCC BAA 2113, and NCTC 13437).

Techniques: In Vivo, Infection

H. pylori transcriptionally increased gastric epithelial NFIL3 expression via ERK-SP1 pathway. ( A ) AGS cells were subjected to infection with H. pylori 11,637 or 26,695 (MOI = 100, 24 h) either in the same (lower) chamber or in a separate (upper) chamber of a Transwell apparatus. The expression levels of NFIL3 were assessed using qRT-PCR ( n = 3) and WB. ( B ) The expression of NFIL3 in AGS cells infected with H. pylori TN2GF4 or its virulence mutant strains was evaluated through the GEO database ( GSE60661 ). ( C ) NFIL3 expression was further examined in AGS cells infected with H. pylori 11,637 or the Δ cagA mutant, utilizing qRT-PCR ( n = 3) and WB. ( D ) The expression of NFIL3 was also analyzed in AGS cells infected with H. pylori 26,695 or the Δ vacA mutant through qRT-PCR ( n = 3) and WB. ( E ) The expression levels of NR1D1 were investigated in AGS cells infected with H. pylori TN2GF4 or virulence mutant strains utilizing the GEO database ( GSE60661 ). ( F ) Potential transcription factor binding sites were predicted via the PROMO website, focusing on a 2000 bp segment of the NFIL3 promoter. ( G ) A conserved sequence of putative SP1 binding sites (red line) was identified in both human and mouse models. ( H ) WB and qRT-PCR ( n = 3) analyses of NFIL3 were conducted in AGS cells post-treatment with H. pylori 11,637, U0126, and Mithramycin A (MMA). ( I ) Following a 24-hour transfection with SP1 siRNA, AGS cells were subsequently infected with H. pylori 11,637 for an additional 24 h. The expression levels of NFIL3 and SP1 were analyzed using qRT-PCR ( n = 3) and WB. ( J ) NFIL3 promoter luciferase reporter assays were conducted in AGS cell lines treated with H. pylori 11,637, U0126, Mithramycin A (MMA), and the Δ cagA mutant. ( K ) Luciferase reporter assay for NFIL3-Luc and mutant-Luc. n.s. P > 0.05, ** P < 0.01, and *** P < 0.001

Journal: Cell Communication and Signaling : CCS

Article Title: Disruption of the biorhythm in gastric epithelial cell triggers inflammation in Helicobacter pylori -associated gastritis by aberrantly regulating NFIL3 via CagA activated ERK-SP1 pathway

doi: 10.1186/s12964-025-02302-z

Figure Lengend Snippet: H. pylori transcriptionally increased gastric epithelial NFIL3 expression via ERK-SP1 pathway. ( A ) AGS cells were subjected to infection with H. pylori 11,637 or 26,695 (MOI = 100, 24 h) either in the same (lower) chamber or in a separate (upper) chamber of a Transwell apparatus. The expression levels of NFIL3 were assessed using qRT-PCR ( n = 3) and WB. ( B ) The expression of NFIL3 in AGS cells infected with H. pylori TN2GF4 or its virulence mutant strains was evaluated through the GEO database ( GSE60661 ). ( C ) NFIL3 expression was further examined in AGS cells infected with H. pylori 11,637 or the Δ cagA mutant, utilizing qRT-PCR ( n = 3) and WB. ( D ) The expression of NFIL3 was also analyzed in AGS cells infected with H. pylori 26,695 or the Δ vacA mutant through qRT-PCR ( n = 3) and WB. ( E ) The expression levels of NR1D1 were investigated in AGS cells infected with H. pylori TN2GF4 or virulence mutant strains utilizing the GEO database ( GSE60661 ). ( F ) Potential transcription factor binding sites were predicted via the PROMO website, focusing on a 2000 bp segment of the NFIL3 promoter. ( G ) A conserved sequence of putative SP1 binding sites (red line) was identified in both human and mouse models. ( H ) WB and qRT-PCR ( n = 3) analyses of NFIL3 were conducted in AGS cells post-treatment with H. pylori 11,637, U0126, and Mithramycin A (MMA). ( I ) Following a 24-hour transfection with SP1 siRNA, AGS cells were subsequently infected with H. pylori 11,637 for an additional 24 h. The expression levels of NFIL3 and SP1 were analyzed using qRT-PCR ( n = 3) and WB. ( J ) NFIL3 promoter luciferase reporter assays were conducted in AGS cell lines treated with H. pylori 11,637, U0126, Mithramycin A (MMA), and the Δ cagA mutant. ( K ) Luciferase reporter assay for NFIL3-Luc and mutant-Luc. n.s. P > 0.05, ** P < 0.01, and *** P < 0.001

Article Snippet: We utilized several strains of H. pylori , specifically the cagA and vacA -positive strain 26,695 (ATCC 700392), the vacA knockout mutant strain 26,695 (referred to as Δ vacA herein), the cagA and vacA -positive strain NCTC 11,637 (ATCC 43504, designated as H. pylori 11637 in this manuscript), the cagA knockout mutant strain NCTC 11,637 (termed Δ cagA in this article), and the cagA and vacA -positive strain PMSS1 (pre-mouse Sydney strain 1) [ , ].

Techniques: Expressing, Infection, Quantitative RT-PCR, Mutagenesis, Binding Assay, Sequencing, Transfection, Luciferase, Reporter Assay

 A. hominis  isolates examined in this study ( n = 46)

Journal:

Article Title: Clinical Significance and Taxonomy of Actinobacillus hominis

doi: 10.1128/JCM.39.3.930-935.2001

Figure Lengend Snippet: A. hominis isolates examined in this study ( n = 46)

Article Snippet: The biochemical reactions of the A. hominis strains are shown together with reactions of the 12 type and reference strains of the closely related taxa (Table ). table ft1 table-wrap mode="anchored" t5 TABLE 4 caption a7 Characteristic Results a with: A. hominis (46 strains) A. capsulatus NCTC 11408 T A. equuli NCTC 8529 T A. lignieresii NCTC 4189 T A. muris CCUG 16938 T A. pleuropneumoniae ATCC 27088 T A. suis CCM 5586 T A. ureae NCTC 10219 T Bisgaard taxon 8 CCUG 16494 Bisgaard taxon 9 CCUG 15571 Bisgaard taxon 11 CCUG 15573 P. pneu- motropica (Jawetz) NCTC 8141 T P. pneumotropica (Heyl) ATCC 12555 Hemolysis 0 0 0 0 + + + 0 0 0 0 + + Oxidase production + + + + + 0 + 0 + + 0 + + Urease production + + + + + + + + + + + + + β-Galactosidase (ONPG) production 45/46 + + + 0 + + 0 + + + + + β-Glucuronidase (PGUA) production 0 0 0 0 + 0 0 0 0 0 0 0 0 Indole production 0 0 0 0 0 0 0 0 0 0 0 + + Nitrate reduction 23/23 + + + + + + + + + + + + Nitrite reduction 23/23 0 + + 0 + + 0 + + + 0 + Gelatinase production 0 0 0 0 0 0 0 0 0 0 0 0 0 Arginine production 0 0 0 0 + 0 0 0 0 0 0 0 0 Lysine production 0 0 0 0 0 0 0 0 0 0 0 0 + Ornithine production 0 0 0 0 0 0 0 0 0 0 0 + + Acid production l -Arabinose 0 + 0 0 0 0 + 0 0 0 0 0 + d -Xylose + + + + 0 + + 0 + + + + + l -Rhamnose 0 0 0 0 0 0 0 0 0 0 + 0 0 d -Glucose b + + + + + + + + + + + + + d -Galactose + + + + 0 0 + 0 0 + + + + d -Mannose 9/46 + + + + + + 0 + + + + + Sucrose + + + + + + + + + + + + + Lactose 45/46 + + + 0 0 + 0 + + + 0 + Maltose 45/46 + + + + + + + + + + + + Cellobiose 2/46 + 0 0 + 0 + 0 0 0 + 0 0 Trehalose + + + 0 + 0 + 0 0 + + + + Melibiose 44/46 + + 0 + 0 + 0 + + + 0 + Adonitol 0 0 0 0 0 0 0 0 0 0 0 0 0 Dulcitol 0 0 0 0 0 0 0 0 0 0 0 0 0 d -Sorbitol 0 + 0 0 0 0 0 0 0 0 0 0 0 d -Mannitol + + + + + + 0 + + + 0 0 0 Inositol 0 0 0 0 0 0 0 0 0 0 0 + 0 Salicin 23/46 + 0 0 + + + 0 0 0 + 0 0 Esculin 15/46 + 0 0 + 0 + 0 0 0 + 0 0 Raffinose + + + 0 0 0 + 0 + + + 0 + Open in a separate window a 0, negative; +, positive. b No gas produced from glucose by any strains.

Techniques: Isolation

 A. hominis  -related taxa examined in this study ( n = 20)

Journal:

Article Title: Clinical Significance and Taxonomy of Actinobacillus hominis

doi: 10.1128/JCM.39.3.930-935.2001

Figure Lengend Snippet: A. hominis -related taxa examined in this study ( n = 20)

Article Snippet: The biochemical reactions of the A. hominis strains are shown together with reactions of the 12 type and reference strains of the closely related taxa (Table ). table ft1 table-wrap mode="anchored" t5 TABLE 4 caption a7 Characteristic Results a with: A. hominis (46 strains) A. capsulatus NCTC 11408 T A. equuli NCTC 8529 T A. lignieresii NCTC 4189 T A. muris CCUG 16938 T A. pleuropneumoniae ATCC 27088 T A. suis CCM 5586 T A. ureae NCTC 10219 T Bisgaard taxon 8 CCUG 16494 Bisgaard taxon 9 CCUG 15571 Bisgaard taxon 11 CCUG 15573 P. pneu- motropica (Jawetz) NCTC 8141 T P. pneumotropica (Heyl) ATCC 12555 Hemolysis 0 0 0 0 + + + 0 0 0 0 + + Oxidase production + + + + + 0 + 0 + + 0 + + Urease production + + + + + + + + + + + + + β-Galactosidase (ONPG) production 45/46 + + + 0 + + 0 + + + + + β-Glucuronidase (PGUA) production 0 0 0 0 + 0 0 0 0 0 0 0 0 Indole production 0 0 0 0 0 0 0 0 0 0 0 + + Nitrate reduction 23/23 + + + + + + + + + + + + Nitrite reduction 23/23 0 + + 0 + + 0 + + + 0 + Gelatinase production 0 0 0 0 0 0 0 0 0 0 0 0 0 Arginine production 0 0 0 0 + 0 0 0 0 0 0 0 0 Lysine production 0 0 0 0 0 0 0 0 0 0 0 0 + Ornithine production 0 0 0 0 0 0 0 0 0 0 0 + + Acid production l -Arabinose 0 + 0 0 0 0 + 0 0 0 0 0 + d -Xylose + + + + 0 + + 0 + + + + + l -Rhamnose 0 0 0 0 0 0 0 0 0 0 + 0 0 d -Glucose b + + + + + + + + + + + + + d -Galactose + + + + 0 0 + 0 0 + + + + d -Mannose 9/46 + + + + + + 0 + + + + + Sucrose + + + + + + + + + + + + + Lactose 45/46 + + + 0 0 + 0 + + + 0 + Maltose 45/46 + + + + + + + + + + + + Cellobiose 2/46 + 0 0 + 0 + 0 0 0 + 0 0 Trehalose + + + 0 + 0 + 0 0 + + + + Melibiose 44/46 + + 0 + 0 + 0 + + + 0 + Adonitol 0 0 0 0 0 0 0 0 0 0 0 0 0 Dulcitol 0 0 0 0 0 0 0 0 0 0 0 0 0 d -Sorbitol 0 + 0 0 0 0 0 0 0 0 0 0 0 d -Mannitol + + + + + + 0 + + + 0 0 0 Inositol 0 0 0 0 0 0 0 0 0 0 0 + 0 Salicin 23/46 + 0 0 + + + 0 0 0 + 0 0 Esculin 15/46 + 0 0 + 0 + 0 0 0 + 0 0 Raffinose + + + 0 0 0 + 0 + + + 0 + Open in a separate window a 0, negative; +, positive. b No gas produced from glucose by any strains.

Techniques:

Underlying diseases and other predisposing factors in 36 patients with  A. hominis  infection a

Journal:

Article Title: Clinical Significance and Taxonomy of Actinobacillus hominis

doi: 10.1128/JCM.39.3.930-935.2001

Figure Lengend Snippet: Underlying diseases and other predisposing factors in 36 patients with A. hominis infection a

Article Snippet: The biochemical reactions of the A. hominis strains are shown together with reactions of the 12 type and reference strains of the closely related taxa (Table ). table ft1 table-wrap mode="anchored" t5 TABLE 4 caption a7 Characteristic Results a with: A. hominis (46 strains) A. capsulatus NCTC 11408 T A. equuli NCTC 8529 T A. lignieresii NCTC 4189 T A. muris CCUG 16938 T A. pleuropneumoniae ATCC 27088 T A. suis CCM 5586 T A. ureae NCTC 10219 T Bisgaard taxon 8 CCUG 16494 Bisgaard taxon 9 CCUG 15571 Bisgaard taxon 11 CCUG 15573 P. pneu- motropica (Jawetz) NCTC 8141 T P. pneumotropica (Heyl) ATCC 12555 Hemolysis 0 0 0 0 + + + 0 0 0 0 + + Oxidase production + + + + + 0 + 0 + + 0 + + Urease production + + + + + + + + + + + + + β-Galactosidase (ONPG) production 45/46 + + + 0 + + 0 + + + + + β-Glucuronidase (PGUA) production 0 0 0 0 + 0 0 0 0 0 0 0 0 Indole production 0 0 0 0 0 0 0 0 0 0 0 + + Nitrate reduction 23/23 + + + + + + + + + + + + Nitrite reduction 23/23 0 + + 0 + + 0 + + + 0 + Gelatinase production 0 0 0 0 0 0 0 0 0 0 0 0 0 Arginine production 0 0 0 0 + 0 0 0 0 0 0 0 0 Lysine production 0 0 0 0 0 0 0 0 0 0 0 0 + Ornithine production 0 0 0 0 0 0 0 0 0 0 0 + + Acid production l -Arabinose 0 + 0 0 0 0 + 0 0 0 0 0 + d -Xylose + + + + 0 + + 0 + + + + + l -Rhamnose 0 0 0 0 0 0 0 0 0 0 + 0 0 d -Glucose b + + + + + + + + + + + + + d -Galactose + + + + 0 0 + 0 0 + + + + d -Mannose 9/46 + + + + + + 0 + + + + + Sucrose + + + + + + + + + + + + + Lactose 45/46 + + + 0 0 + 0 + + + 0 + Maltose 45/46 + + + + + + + + + + + + Cellobiose 2/46 + 0 0 + 0 + 0 0 0 + 0 0 Trehalose + + + 0 + 0 + 0 0 + + + + Melibiose 44/46 + + 0 + 0 + 0 + + + 0 + Adonitol 0 0 0 0 0 0 0 0 0 0 0 0 0 Dulcitol 0 0 0 0 0 0 0 0 0 0 0 0 0 d -Sorbitol 0 + 0 0 0 0 0 0 0 0 0 0 0 d -Mannitol + + + + + + 0 + + + 0 0 0 Inositol 0 0 0 0 0 0 0 0 0 0 0 + 0 Salicin 23/46 + 0 0 + + + 0 0 0 + 0 0 Esculin 15/46 + 0 0 + 0 + 0 0 0 + 0 0 Raffinose + + + 0 0 0 + 0 + + + 0 + Open in a separate window a 0, negative; +, positive. b No gas produced from glucose by any strains.

Techniques: Infection

Characteristics of  A. hominis  and type and reference strains of related taxa

Journal:

Article Title: Clinical Significance and Taxonomy of Actinobacillus hominis

doi: 10.1128/JCM.39.3.930-935.2001

Figure Lengend Snippet: Characteristics of A. hominis and type and reference strains of related taxa

Article Snippet: The biochemical reactions of the A. hominis strains are shown together with reactions of the 12 type and reference strains of the closely related taxa (Table ). table ft1 table-wrap mode="anchored" t5 TABLE 4 caption a7 Characteristic Results a with: A. hominis (46 strains) A. capsulatus NCTC 11408 T A. equuli NCTC 8529 T A. lignieresii NCTC 4189 T A. muris CCUG 16938 T A. pleuropneumoniae ATCC 27088 T A. suis CCM 5586 T A. ureae NCTC 10219 T Bisgaard taxon 8 CCUG 16494 Bisgaard taxon 9 CCUG 15571 Bisgaard taxon 11 CCUG 15573 P. pneu- motropica (Jawetz) NCTC 8141 T P. pneumotropica (Heyl) ATCC 12555 Hemolysis 0 0 0 0 + + + 0 0 0 0 + + Oxidase production + + + + + 0 + 0 + + 0 + + Urease production + + + + + + + + + + + + + β-Galactosidase (ONPG) production 45/46 + + + 0 + + 0 + + + + + β-Glucuronidase (PGUA) production 0 0 0 0 + 0 0 0 0 0 0 0 0 Indole production 0 0 0 0 0 0 0 0 0 0 0 + + Nitrate reduction 23/23 + + + + + + + + + + + + Nitrite reduction 23/23 0 + + 0 + + 0 + + + 0 + Gelatinase production 0 0 0 0 0 0 0 0 0 0 0 0 0 Arginine production 0 0 0 0 + 0 0 0 0 0 0 0 0 Lysine production 0 0 0 0 0 0 0 0 0 0 0 0 + Ornithine production 0 0 0 0 0 0 0 0 0 0 0 + + Acid production l -Arabinose 0 + 0 0 0 0 + 0 0 0 0 0 + d -Xylose + + + + 0 + + 0 + + + + + l -Rhamnose 0 0 0 0 0 0 0 0 0 0 + 0 0 d -Glucose b + + + + + + + + + + + + + d -Galactose + + + + 0 0 + 0 0 + + + + d -Mannose 9/46 + + + + + + 0 + + + + + Sucrose + + + + + + + + + + + + + Lactose 45/46 + + + 0 0 + 0 + + + 0 + Maltose 45/46 + + + + + + + + + + + + Cellobiose 2/46 + 0 0 + 0 + 0 0 0 + 0 0 Trehalose + + + 0 + 0 + 0 0 + + + + Melibiose 44/46 + + 0 + 0 + 0 + + + 0 + Adonitol 0 0 0 0 0 0 0 0 0 0 0 0 0 Dulcitol 0 0 0 0 0 0 0 0 0 0 0 0 0 d -Sorbitol 0 + 0 0 0 0 0 0 0 0 0 0 0 d -Mannitol + + + + + + 0 + + + 0 0 0 Inositol 0 0 0 0 0 0 0 0 0 0 0 + 0 Salicin 23/46 + 0 0 + + + 0 0 0 + 0 0 Esculin 15/46 + 0 0 + 0 + 0 0 0 + 0 0 Raffinose + + + 0 0 0 + 0 + + + 0 + Open in a separate window a 0, negative; +, positive. b No gas produced from glucose by any strains.

Techniques:

Relatedness of the type strain of  A. hominis  and related organisms

Journal:

Article Title: Clinical Significance and Taxonomy of Actinobacillus hominis

doi: 10.1128/JCM.39.3.930-935.2001

Figure Lengend Snippet: Relatedness of the type strain of A. hominis and related organisms

Article Snippet: The biochemical reactions of the A. hominis strains are shown together with reactions of the 12 type and reference strains of the closely related taxa (Table ). table ft1 table-wrap mode="anchored" t5 TABLE 4 caption a7 Characteristic Results a with: A. hominis (46 strains) A. capsulatus NCTC 11408 T A. equuli NCTC 8529 T A. lignieresii NCTC 4189 T A. muris CCUG 16938 T A. pleuropneumoniae ATCC 27088 T A. suis CCM 5586 T A. ureae NCTC 10219 T Bisgaard taxon 8 CCUG 16494 Bisgaard taxon 9 CCUG 15571 Bisgaard taxon 11 CCUG 15573 P. pneu- motropica (Jawetz) NCTC 8141 T P. pneumotropica (Heyl) ATCC 12555 Hemolysis 0 0 0 0 + + + 0 0 0 0 + + Oxidase production + + + + + 0 + 0 + + 0 + + Urease production + + + + + + + + + + + + + β-Galactosidase (ONPG) production 45/46 + + + 0 + + 0 + + + + + β-Glucuronidase (PGUA) production 0 0 0 0 + 0 0 0 0 0 0 0 0 Indole production 0 0 0 0 0 0 0 0 0 0 0 + + Nitrate reduction 23/23 + + + + + + + + + + + + Nitrite reduction 23/23 0 + + 0 + + 0 + + + 0 + Gelatinase production 0 0 0 0 0 0 0 0 0 0 0 0 0 Arginine production 0 0 0 0 + 0 0 0 0 0 0 0 0 Lysine production 0 0 0 0 0 0 0 0 0 0 0 0 + Ornithine production 0 0 0 0 0 0 0 0 0 0 0 + + Acid production l -Arabinose 0 + 0 0 0 0 + 0 0 0 0 0 + d -Xylose + + + + 0 + + 0 + + + + + l -Rhamnose 0 0 0 0 0 0 0 0 0 0 + 0 0 d -Glucose b + + + + + + + + + + + + + d -Galactose + + + + 0 0 + 0 0 + + + + d -Mannose 9/46 + + + + + + 0 + + + + + Sucrose + + + + + + + + + + + + + Lactose 45/46 + + + 0 0 + 0 + + + 0 + Maltose 45/46 + + + + + + + + + + + + Cellobiose 2/46 + 0 0 + 0 + 0 0 0 + 0 0 Trehalose + + + 0 + 0 + 0 0 + + + + Melibiose 44/46 + + 0 + 0 + 0 + + + 0 + Adonitol 0 0 0 0 0 0 0 0 0 0 0 0 0 Dulcitol 0 0 0 0 0 0 0 0 0 0 0 0 0 d -Sorbitol 0 + 0 0 0 0 0 0 0 0 0 0 0 d -Mannitol + + + + + + 0 + + + 0 0 0 Inositol 0 0 0 0 0 0 0 0 0 0 0 + 0 Salicin 23/46 + 0 0 + + + 0 0 0 + 0 0 Esculin 15/46 + 0 0 + 0 + 0 0 0 + 0 0 Raffinose + + + 0 0 0 + 0 + + + 0 + Open in a separate window a 0, negative; +, positive. b No gas produced from glucose by any strains.

Techniques: